To mediate ADCC in vitro [9, 17, 23]. Although the contribution of ADCC activity to clinical efficacy is unclear, it is important to characterize all activities of the candidate mAb, especially those that can be affected by differences in post-translational modifications, such as glycosylation. The ability of ABP 501 to induce ADCC was assessed using MT-3 cells as target cells, and NK-92M1 cells s

Distinction within prokaryotes, formingthe primary taxonomic division within them, which is supported by both molecular sequence data and morphological features, is of the monoderm prokaryotes (Monodermata, i.e., those bounded by a single cell membrane) and the diderm prokaryotes (Didermata, i.e., those bounded by inner and outer cell membranes defining a periplasmic compartment). In that sense, b

Ithout malignancy [53].Non anti-infectious agentsG-CSF and GM-CSFHaemopoietic growth factors, such as granulocyte colonystimulating factor (G-CSF) and granulocyte-macrophage colony-stimulating factor (GM-CSF) have been assessed in several clinical trials [54,55]. The known effect of G-CSF and GM-CSF in increasing the number of circulating neutrophil granulocytes was the rationale for clinical stud

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To mediate ADCC in vitro [9, 17, 23]. Although the contribution of ADCC activity to clinical efficacy is unclear, it is important to characterize all activities of the candidate mAb, especially those that can be affected by differences in post-translational modifications, such as glycosylation. The ability of ABP 501 to induce ADCC was assessed using MT-3 cells as target cells, and NK-92M1 cells s

E bradykinin. The nonapeptide has a very short half-life (a matter of seconds) and exhibits its functions via the B1 and B2 receptors (3). Generating other mediators such as nitric oxide, prostaglandins, and leukotrienes, bradykinin is involved in the regulation of blood pressure, the induction of fever and pain, vascular leakage, and the chemotaxis of immune cells (4). In addition, further proces

Ersus host disease, or other abdominal syndromes including cholecystitis, cholangitis, appendicitis need to be ruled out. The management of neutropenic enterocolitis has evolved over the years as clinical experience has grown. Recent studies have reported the success of conservative treatment in most patients. Surgical intervention is now reserved for selected cases of neutropenic enterocolitis ba

Ersus host disease, or other abdominal syndromes including cholecystitis, cholangitis, appendicitis need to be ruled out. The management of neutropenic enterocolitis has evolved over the years as clinical experience has grown. Recent studies have reported the success of conservative treatment in most patients. Surgical intervention is now reserved for selected cases of neutropenic enterocolitis ba